Which AV block is Second Degree Heart Block Type 1 (Wenckebach), with causes including anoxia, edema after open‑heart surgery, digoxin toxicity, hyperkalemia, and anterior MI?

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Multiple Choice

Which AV block is Second Degree Heart Block Type 1 (Wenckebach), with causes including anoxia, edema after open‑heart surgery, digoxin toxicity, hyperkalemia, and anterior MI?

Wenckebach is an AV nodal block pattern where conduction through the AV node slows progressively with each beat, until a QRS is dropped, then the cycle repeats. The ECG hallmark is a gradually lengthening PR interval followed by a nonconducted beat, after which the PR interval resets and the pattern starts again. This reflects a reversible, nodal-level delay rather than a block below the AV node.

The causes listed fit this AV nodal mechanism because anything that increases vagal tone or directly depresses AV nodal conduction can produce Wenckebach: hypoxia or edema around the heart after surgery can enlarge vagal influence or compress the node; digoxin toxicity directly slows AV nodal conduction; electrolyte disturbances such as hyperkalemia can also slow nodal tissue. Anterior myocardial infarction, however, more often injures tissue below the AV node (the His-Purkinje system), leading to Mobitz type II or higher-grade blocks rather than Wenckebach, which helps distinguish it from other AV blocks.

For contrast, first-degree block shows simply a longer PR interval without dropped beats; second-degree type II shows dropped beats with a relatively constant PR interval rather than progressively lengthening; third-degree block shows complete AV dissociation with no fixed relationship between P waves and QRS complexes.

Thus, the description corresponds to Second Degree Heart Block Type I (Wenckebach).

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